pbabe puro empty vector control (Addgene inc)
96
Structured Review
Addgene inc
pbabe puro empty vector control
Pbabe Puro Empty Vector Control, supplied by Addgene inc, used in various techniques. Bioz Stars score: 96/100, based on 581 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/pbabe+puro+empty+vector+control/pBABE-puro+(Plasmid+%231764)/10__1158_slash_2767___9764__crc___23___0379-57-43-47
Average 96 stars, based on 581 article reviews
Pbabe Puro Empty Vector Control, supplied by Addgene inc, used in various techniques. Bioz Stars score: 96/100, based on 581 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/pbabe+puro+empty+vector+control/pBABE-puro+(Plasmid+%231764)/10__1158_slash_2767___9764__crc___23___0379-57-43-47
Average 96 stars, based on 581 article reviews
pbabe puro empty vector control - by Bioz Stars,
2026-10
96/100 stars
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Expressing:Article Title: PP2A-activating Drugs Enhance FLT3 Inhibitor Efficacy through AKT Inhibition–Dependent GSK-3β–Mediated c-Myc and Pim-1 Proteasomal Degradation Article Snippet: .. The myc-estrogen receptor (ER) expression vector pBABE-puromyc-ER (plasmid No. 19128; ref. 21) and Article Title: Pim kinase inhibitor co-treatment decreases alternative non-homologous end-joining DNA repair and genomic instability induced by topoisomerase 2 inhibitors in cells with FLT3 internal tandem duplication Article Snippet: Ba/F3-ITD cells were infected with pMSCVpuro-Flag-cMyc-T58A retroviral plasmid (Addgene plasmid #20076; https://www.addgene.org/20076 ) [ ]) from Addgene (Cambridge, MA, USA), containing c-Myc with a point mutation at position 58, changing threonine to alanine and thereby inhibiting phosphorylation, or pMSCVpuro empty vector control (Takara Bio USA, Mountain View, CA, USA). .. To induce c-Myc overexpression, Ba/F3-ITD cells were infected with the c-Myc expression vector pBABEpuro-myc-ER (estrogen receptor) (plasmid #19128; https://www.addgene.org/19128 ) [ ] or Plasmid Preparation:Article Title: PP2A-activating Drugs Enhance FLT3 Inhibitor Efficacy through AKT Inhibition–Dependent GSK-3β–Mediated c-Myc and Pim-1 Proteasomal Degradation Article Snippet: .. The myc-estrogen receptor (ER) expression vector pBABE-puromyc-ER (plasmid No. 19128; ref. 21) and Article Title: Pim Kinase Inhibitors Increase Gilteritinib Cytotoxicity in FLT3-ITD Acute Myeloid Leukemia Through GSK-3β Activation and c-Myc and Mcl-1 Proteasomal Degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1-S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372 ) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation , pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371 ), containing wild-type Mcl-1 , or Article Title: Pim kinase inhibitors increase gilteritinib cytotoxicity in FLT3-ITD acute myeloid leukemia through GSK-3β activation and c-Myc and Mcl-1 proteasomal degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation (27), pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371), containing wild-type Mcl-1 (27), or Article Title: Pim kinase inhibitor co-treatment decreases alternative non-homologous end-joining DNA repair and genomic instability induced by topoisomerase 2 inhibitors in cells with FLT3 internal tandem duplication Article Snippet: Ba/F3-ITD cells were infected with pMSCVpuro-Flag-cMyc-T58A retroviral plasmid (Addgene plasmid #20076; https://www.addgene.org/20076 ) [ ]) from Addgene (Cambridge, MA, USA), containing c-Myc with a point mutation at position 58, changing threonine to alanine and thereby inhibiting phosphorylation, or pMSCVpuro empty vector control (Takara Bio USA, Mountain View, CA, USA). .. To induce c-Myc overexpression, Ba/F3-ITD cells were infected with the c-Myc expression vector pBABEpuro-myc-ER (estrogen receptor) (plasmid #19128; https://www.addgene.org/19128 ) [ ] or Control:Article Title: PP2A-activating Drugs Enhance FLT3 Inhibitor Efficacy through AKT Inhibition–Dependent GSK-3β–Mediated c-Myc and Pim-1 Proteasomal Degradation Article Snippet: .. The myc-estrogen receptor (ER) expression vector pBABE-puromyc-ER (plasmid No. 19128; ref. 21) and Article Title: Pim Kinase Inhibitors Increase Gilteritinib Cytotoxicity in FLT3-ITD Acute Myeloid Leukemia Through GSK-3β Activation and c-Myc and Mcl-1 Proteasomal Degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1-S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372 ) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation , pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371 ), containing wild-type Mcl-1 , or Article Title: Pim kinase inhibitors increase gilteritinib cytotoxicity in FLT3-ITD acute myeloid leukemia through GSK-3β activation and c-Myc and Mcl-1 proteasomal degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation (27), pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371), containing wild-type Mcl-1 (27), or Article Title: Pim kinase inhibitor co-treatment decreases alternative non-homologous end-joining DNA repair and genomic instability induced by topoisomerase 2 inhibitors in cells with FLT3 internal tandem duplication Article Snippet: Ba/F3-ITD cells were infected with pMSCVpuro-Flag-cMyc-T58A retroviral plasmid (Addgene plasmid #20076; https://www.addgene.org/20076 ) [ ]) from Addgene (Cambridge, MA, USA), containing c-Myc with a point mutation at position 58, changing threonine to alanine and thereby inhibiting phosphorylation, or pMSCVpuro empty vector control (Takara Bio USA, Mountain View, CA, USA). .. To induce c-Myc overexpression, Ba/F3-ITD cells were infected with the c-Myc expression vector pBABEpuro-myc-ER (estrogen receptor) (plasmid #19128; https://www.addgene.org/19128 ) [ ] or Retroviral:Article Title: Pim Kinase Inhibitors Increase Gilteritinib Cytotoxicity in FLT3-ITD Acute Myeloid Leukemia Through GSK-3β Activation and c-Myc and Mcl-1 Proteasomal Degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1-S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372 ) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation , pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371 ), containing wild-type Mcl-1 , or Article Title: Pim kinase inhibitors increase gilteritinib cytotoxicity in FLT3-ITD acute myeloid leukemia through GSK-3β activation and c-Myc and Mcl-1 proteasomal degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation (27), pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371), containing wild-type Mcl-1 (27), or Infection:Article Title: Pim Kinase Inhibitors Increase Gilteritinib Cytotoxicity in FLT3-ITD Acute Myeloid Leukemia Through GSK-3β Activation and c-Myc and Mcl-1 Proteasomal Degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1-S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372 ) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation , pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371 ), containing wild-type Mcl-1 , or Article Title: Pim kinase inhibitors increase gilteritinib cytotoxicity in FLT3-ITD acute myeloid leukemia through GSK-3β activation and c-Myc and Mcl-1 proteasomal degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation (27), pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371), containing wild-type Mcl-1 (27), or Article Title: Pim kinase inhibitor co-treatment decreases alternative non-homologous end-joining DNA repair and genomic instability induced by topoisomerase 2 inhibitors in cells with FLT3 internal tandem duplication Article Snippet: Ba/F3-ITD cells were infected with pMSCVpuro-Flag-cMyc-T58A retroviral plasmid (Addgene plasmid #20076; https://www.addgene.org/20076 ) [ ]) from Addgene (Cambridge, MA, USA), containing c-Myc with a point mutation at position 58, changing threonine to alanine and thereby inhibiting phosphorylation, or pMSCVpuro empty vector control (Takara Bio USA, Mountain View, CA, USA). .. To induce c-Myc overexpression, Ba/F3-ITD cells were infected with the c-Myc expression vector pBABEpuro-myc-ER (estrogen receptor) (plasmid #19128; https://www.addgene.org/19128 ) [ ] or Mutagenesis:Article Title: Pim Kinase Inhibitors Increase Gilteritinib Cytotoxicity in FLT3-ITD Acute Myeloid Leukemia Through GSK-3β Activation and c-Myc and Mcl-1 Proteasomal Degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1-S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372 ) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation , pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371 ), containing wild-type Mcl-1 , or Article Title: Pim kinase inhibitors increase gilteritinib cytotoxicity in FLT3-ITD acute myeloid leukemia through GSK-3β activation and c-Myc and Mcl-1 proteasomal degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation (27), pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371), containing wild-type Mcl-1 (27), or Residue:Article Title: Pim Kinase Inhibitors Increase Gilteritinib Cytotoxicity in FLT3-ITD Acute Myeloid Leukemia Through GSK-3β Activation and c-Myc and Mcl-1 Proteasomal Degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1-S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372 ) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation , pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371 ), containing wild-type Mcl-1 , or Article Title: Pim kinase inhibitors increase gilteritinib cytotoxicity in FLT3-ITD acute myeloid leukemia through GSK-3β activation and c-Myc and Mcl-1 proteasomal degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation (27), pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371), containing wild-type Mcl-1 (27), or Phospho-proteomics:Article Title: Pim Kinase Inhibitors Increase Gilteritinib Cytotoxicity in FLT3-ITD Acute Myeloid Leukemia Through GSK-3β Activation and c-Myc and Mcl-1 Proteasomal Degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1-S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372 ) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation , pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371 ), containing wild-type Mcl-1 , or Article Title: Pim kinase inhibitors increase gilteritinib cytotoxicity in FLT3-ITD acute myeloid leukemia through GSK-3β activation and c-Myc and Mcl-1 proteasomal degradation Article Snippet: .. Ba/F3-ITD cells also underwent retroviral infection with pBabe-Flag hMcl-1S159A plasmid (Addgene plasmid # 25372; RRID:Addgene_25372) containing Mcl-1 with a mutation changing serine to alanine at residue 159, preventing phosphorylation (27), pBabe-Flag hMcl-1 plasmid (Addgene plasmid # 25371; RRID:Addgene_25371), containing wild-type Mcl-1 (27), or Over Expression:Article Title: Pim kinase inhibitor co-treatment decreases alternative non-homologous end-joining DNA repair and genomic instability induced by topoisomerase 2 inhibitors in cells with FLT3 internal tandem duplication Article Snippet: Ba/F3-ITD cells were infected with pMSCVpuro-Flag-cMyc-T58A retroviral plasmid (Addgene plasmid #20076; https://www.addgene.org/20076 ) [ ]) from Addgene (Cambridge, MA, USA), containing c-Myc with a point mutation at position 58, changing threonine to alanine and thereby inhibiting phosphorylation, or pMSCVpuro empty vector control (Takara Bio USA, Mountain View, CA, USA). .. To induce c-Myc overexpression, Ba/F3-ITD cells were infected with the c-Myc expression vector pBABEpuro-myc-ER (estrogen receptor) (plasmid #19128; https://www.addgene.org/19128 ) [ ] or |